The luminal AAA+ ATPase torsinA mediates distinct mechanisms of  nuclear-cytoplasmic communication by adopting different functional assembly  states

The luminal AAA+ ATPase torsinA mediates distinct mechanisms of nuclear-cytoplasmic communication by adopting different functional assembly states

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Torsin ATPases influence chromatin interaction of the Torsin regulator LAP1

Figure 2 from The LINC and NPC relationship – it's complicated!

Observation of a Transient Reaction Intermediate Illuminates the Mechanochemical Cycle of the AAA-ATPase p97

Torsin ATPases influence chromatin interaction of the Torsin regulator LAP1

Aberrant Cellular Behavior of Mutant TorsinA Implicates Nuclear Envelope Dysfunction in DYT1 Dystonia

Torsin ATPases influence chromatin interaction of the Torsin regulator LAP1

LINC Complexes Form by Binding of Three KASH Peptides to Domain Interfaces of Trimeric SUN Proteins: Cell

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Aberrant Cellular Behavior of Mutant TorsinA Implicates Nuclear Envelope Dysfunction in DYT1 Dystonia

The mechanism of Torsin ATPase activation

Disruption of Protein Processing in the Endoplasmic Reticulum of DYT1 Knock-in Mice Implicates Novel Pathways in Dystonia Pathogenesis

Frontiers Torsin ATPases: Harnessing Dynamic Instability for Function

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